|
Image from Baker Hughes. |
Baker Hughes announced the commercial release of FATHOM XT SUBSEA525 inhibitor that helps control asphaltene deposition in deepwater wells.
The inhibitor was designed and certified for offshore applications using a proprietary qualification protocol and a stringent laboratory evaluation method to enable full compatibility with subsea equipment and effective performance at low treatment levels.
During production, crude oils can deposit asphaltenes inside pumps and pipes, creating serious production issues such as plugged flow lines and clogged equipment, resulting in the need to stop operations and perform a costly remediation procedure to get production back online at acceptable levels. Many times, these procedures only offer temporary relief. To lessen the risk of asphaltene deposition and enhance flow, the FATHOM XT inhibitor can be applied at low treatment levels during initial production and throughout the life of the well. The low dosage rate is supposed to simplify supply logistics, reduce onsite storage and lower handling risk.
Baker Hughes tests and qualifies all FATHOM XT chemicals for materials compatibility and reliability using the company’s proprietary 16-test qualification protocol. The test protocol is based on the American Petroleum Institute’s specification standard 17TR6, Attributes of Production Chemicals in Subsea Production Systems. The FATHOM XT SUBSEA525 inhibitor is the first of a new line of high-performance asphaltene inhibitors, which also includes the PAO3086 inhibitor for land-based applications.
This new line of asphaltene inhibitors was developed using the company’s new laboratory evaluation method, the Analytical Centrifuge Stability Analysis for Asphaltenes or ACSAA method. This enables Baker Hughes to evaluate the stability of asphaltenes in various crude oils, and monitor the effectiveness of field treatments.
The FATHOM XT SUBSEA525 asphaltene inhibitor is the latest example of Baker Hughes strategy to improve well efficiency, optimize production and increase ultimate recovery.